Thursday, October 30, 2008
LIGHTENING COLLOIDAL SILVER
Enriching Gifts Internationale's Lightening Colloidal Silver moves in your blood and enters your cells to find and destroy harmful organisms within minutes after contact. The only Colloidal Silver product charged with extremely high lightning voltage instead of 110V or 220V electrical current. One ounce of Lightning Colloidal Silver contains over Nine Billion electrically charged positive ions of Pure Silver material which attach to bacteria, virus, fungus, and germs in your body and neutralize them.
1 oz once a day as needed not to exceed 6 weeks continued use orally. Ideally used on a empty stomach (not recommended for use while taking Pro-Biotic)
KEY BENEFITS:
Silver ions, due to their small molecular size, are the best form for the body because they travel freely in and out of cells
Lightning Colloidal Silver ions are produced using environmental lighting bolts not with 110 or 220 volt current.
PROPERTY BLEND:
Purified deionized micro filtered water
Ten parts per million of Silver mineral (99.999%)
One ounce contains over Nine BILLION electrically charged ions of Pure Silver mineral.
Monday, October 27, 2008
How are Gallstones Diagnosed?
- Computerized tomography (CT) scan - The CT scan is a noninvasive x ray that produces cross-section images of the body. The test may show the gallstones or complications, such as infection and rupture of the gallbladder or bile ducts.
- Cholescintigraphy ( HIDA scan ) - The patient is injected with a small amount of non harmful radioactive material that is absorbed by the gallbladder, which is then stimulated to contract. The test is used to diagnose abnormal contraction of the gallbladder or obstruction of the bile ducts.
- Endoscopic Retrograde Cholangiopancreatography ( ERCP ) - ERCP is used to locate and remove stones in the bile ducts. After lightly sedating you, the doctor inserts an endoscope- a long, flexible, lighted tube with a camera - down the throat and through the stomach and into the small intestine. The endoscope is connected to a computer and video monitor. The endoscope helps the doctor locate the affected bile duct and the gallstone. The stone is captured in a tiny basket and removed with the endoscope.
- Blood tests- Blood tests may be performed to look for signs of infection, obstruction, pancreatitis, or jaundice.
Because gallstone symptoms may be similar to those of a heart attack, appendicitis, ulcers, irritable bowel syndrome, hiatal hernia, pancreatitis, and hepatitis, an accurate diagnosis is important.
(info found @NNDIC/gov/disease/pubs/pdf 145)
Saturday, October 25, 2008
What are the Symptoms of Gallstones?
- steady pain in the right upper abdomen that increases rapidly and lasts from 30 minutes to several hours
- pain in the back between the shoulder blades
- pain under the right shoulder
Notify your doctor if you think you have experienced a gallbladder attack. Although these attacks often pass as gallstones move, your gallbladder can become infected and rupture if a blockage remains.
People with any of the following should see a doctor immediately:
- prolonged pain - more than 5 hours
- nausea and vomiting
- fever -even low grade - or chills
- yellowish color of the skin or whites of the eyes
- clay-colored stools
Many people with gallstones have no symptoms: these gallstones are called "silent stones". They do not interfere with the gallbladder, liver, or pancreas function and do not need treatment.
(information found @NDDIC/gov/disease/pubs PDF 145version)
Friday, October 24, 2008
WHO IS AT RISK FOR GALLSTONES?
- women - especially women who are pregnant, use hormone replacement therapy, or take birth control pills
- people over the age 60
- American Indians
- Mexican Americans
- Overweight or obese men and women
- people who fast or lose a lot of weight quickly
- people with family history of gallstones
- people with diabetes
- people who take cholesterol - lowering drugs
(info found @NDDIC gov/disease/pubs/gallstones/pdf 145)
Thursday, October 23, 2008
SKIN NUTRIENTS
Wednesday, October 22, 2008
MORE ABOUT B VITAMINS
Tuesday, October 21, 2008
IMMUNE SYSTEM
Saturday, October 18, 2008
What Causes Gallstones?
The causes of pigment stones is not fully understood. The stones tend to develop in people who have liver cirrhosis, biliary tract infections, or hereditary blood disorders-such as sickle cell anemia-in which the liver makes too much bilirubin.
The mere presence of gallstones may cause more gallstones to develop. Other factors that contribute to the formation of gallstones, particularly cholesterol stones, include
- Sex- Women are twice likely as men to develop gallstones. Excess estrogen from pregnancy, hormone replacement therapy, and birth control pills appears to increase cholesterol levels in bile and decrease gallbladder movement, which can lead to gallstones.
- Family History - Gallstones often run in families, pointing to a possible genetic link.
- Weight - A large clinical study showed that being even moderately overweight increases the risk for developing gallstones. The most likely reason is that the amount of bile salts in bile is reduced, resulting in more cholesterol. Increased cholesterol reduces gallbladder emptying. Obesity is a major risk factor for gallstones, especially in women.
- Diet - Diets high in fat and cholesterol and low in fiber increases the risk of gallstones due to increased cholesterol in the bile and reduced gallbladder emptying.
- Rapid Weight Loss - As the body metabolizes fat during prolonged fasting and rapid weight loss such as "crash diets" - the liver secretes extra cholesterol into bile, which can cause gallstones. In addition, the gallbladder does not empty properly.
- Ethnicity- American Indians have a genetic predisposition to secrete high levels of cholesterol in bile. In fact, they have the highest rate of gallstones in the United States. The majority of American Indian men have gallstones by age 60. Among the Pima Indians of Arizona, 70 percent of women have gallstones by age 30. Mexican Americans men and women of all ages also have high rates of gallstones.
- Cholesterol-lowering Drugs - Drugs that lower cholesterol levels in the blood actually increase the amount of cholesterol secreted into bile. In turn, the risk of gallstones increases.
- Diabetes - People with diabetes generally have high levels of fatty acids called triglycerides. These fatty acids may increase the risk of gallstones.
(info found NDDIC -PDF version 145-KB gov/disease/pubs)
Friday, October 17, 2008
GALLSTONES
Gallstones are small, pebble-like substances that develop in the gallbladder. The gallbladder is a small pear-shaped sac located below your liver in the right upper abdomen. Gallstones form when liquid stored int the gallbladder hardens into pieces of stone-like material. The liquid- called bile - helps the body digest fats. Bile is made in the liver, then stored in the gallbladder until the body needs it. The gallbladder contracts and pushes the bile into a tube- called the common bile duct- that carries it to the small intestine, where it helps with digestion.
Bile contains water, cholesterol, fats, bile salts, proteins, and bilirubin- a waste product. Bile salts break up fat, and bilirubin gives bile and stool a yellowish-brown color. If the liquid bile contains too much cholesterol, bile salts, or bilirubin, it can harden into gallstones.
The two types of gallstones are cholesterol stones and pigment stones. Cholesterol stones are usually yellow-green and are made primarily of hardened cholesterol. They account for about 80% of gallstones. Pigment stones are small, dark stones made of bilirubin. Gallstones can be as small as a grain of sand or as large as a golf ball. The gallbladder can develop just one large stone, hundreds of tiny stones, or a combination of the two. Gallstones can block normal flow of bile if they move from the gallbladder and lodge in any of the ducts that carry bile from the liver to the small intestine. The ducts include the
- hepatic ducts, which carry bile out of the liver
- cystic duct, which takes bile to and from the gallbladder
- common bile duct, which takes bile from the cystic and hepatic ducts to the small intestine
Bile trapped in these ducts can cause inflammation in the gallbladder, the ducts, or in rare cases, the liver. Other ducts open into the common bile duct, including the pancreatic duct, which carries digestive enzymes out of the pancreas. Sometimes gallstones passing through the common bile duct provoke inflammation in the pancreas- called gallstone pancreatitis- an extremely painful and potentially dangerous condition.
If any of the bile ducts remain blocked for a significant period of time, severe damage or infection can occur in the gallbladder, liver or pancreas. Left untreated, the condition can be fatal. Warning signs of a serious problem are fever, jaundice, and persistent pain.
(NDDIC NIH Publication July 2007)
Tuesday, October 14, 2008
ETIOLOGY
Hypercalciuria is an inherited condition that is the cause of stones in > 50% of affected patients. In these persons, high levels of calcium in the urine cause crystals of calcium oxalate or calcium phosphate to form in the kidneys or urinary tract.
Stones may be more likely to form in a person with a family history of kidney stones. Urinary tract infections, kidney disorders such as cystic kidney diseases, and certain metabolic disorders such as hyperparathyroidism are also linked to stone formation.
Cystinuria and hyperoxaluria are 2 other rare, inherited metabolic disorders that often cause kidney stones. In cystinuria, excessive amounts of the amino acid cystine are excreted in the urine. This can lead to the formation of stones made of cystine. In patients with hyperoxaluria, an excessive quantity of oxalate is produced by the body. This excess is not able to undergo dissolution in the urine, and the crystals settle out and form stones.
Kidney stones develop in more than 70% of persons with a rare hereditary disease called
renal tubular acidosis.
Other causes of kidney stones are hyperuricosuria, a disorder of uric acid metabolism, gout, excess intake of vitamin D, urinary tract infections, and urinary tract obstructions. Certain diuretics based antacids also may in crease the risk of kidney stones by increasing the amount of calcium in the urine.
Calcium oxalate stones may also form in persons who have chronic bowel inflammation or who have undergone intestinal bypass or ostomy surgery. As mentioned previously, struvite stones can form in persons who have had a urinary tract infection. Finally, persons who receive the protease inhibitor indinavir are at risk of having kidney stones.
(information found @family practice news)
Thursday, October 9, 2008
SIGNS & SYMPTOMS
If the stone is too large to pass easily, pain continues as muscles in the wall of the ureter contract in an effort to move the stone into the bladder. As the stone enlarges or travels, blood may appear in the urine. As the stones moves from the ureter toward the bladder, a patient also my experience polyuria or burning with urination.
Fever, Chills, or vomiting may accompany these symptoms
(information found @ family practice news)
Friday, October 3, 2008
KIDNEY STONES -scope-
Kidney stones are one of the most common disorders of the urinary tract. In 2000, 2.7 million visits to the health care providers as well as more than 600,000 visits to the emergency departments for kidney stone problems were documented.
For unknown reasons, the number of persons, in the United States with kidney stones has been increasing over the past 30 years. The prevalence of stone-forming disease rose from 3.8% in the late 1970's to 5.2% in the late 1980's and early 1900's. White Americans are more prone to have kidney stones than are black Americans.
Men tend to be affected with kidney stones more frequently than women. The prevalence of kidney stones rises dramatically as men enter their 40's , and it continues to rise until they are in their 70's. For women, the prevalence of kidney stones peaks in their 50's.
Kidney stones tend to recur in persons who have had at least 1 episode of stone formation.
(information found @ Family Practice News)
Thursday, October 2, 2008
KIDNEY STONES -Etiology-
Hypercalciuria is an inherited condition that is the cause of stones in >50% of affected patients. In these persons, high levels of calcium in the urine cause crystals of calcium oxalate or calcium phosphate to form in the kidneys.
Stones may be more likely to form in a person with a family history of kidney stones. Urinary tract infections, kidney disorders such as cystic kidney diseases, and certain metabolic disorders such as hyperparathyroidism are also linked to stone formation.
Cystinuria and hyperoxaluria are 2 other rare, inherited metabolic disorders that often cause kidney stones. In cystinuria, excessive amounts of the amino acid cystine are excreted in the urine. This can lead to the formation of stones made of cystine. In patients with hyperoxaluria, an excessive quantity of oxalate is produced by the body. This excess is not able to undergo dissolution in the urine, and the crystals settle out and form stones.
Kidney stones develop in more that 70% of persons with a rare hereditary disease called renal tubular acidosis.
Other causes of kidney stones are hyperuricosuria, a disorder of uric acid metabolism, gout, excess intake of Vitamin D, urinary tract infections, and urinary tract obstruction. Certain diuretics or calcium based antacids also may increase risk of kidney stones by increasing the amount of calcium in the urine.
Calcium oxalate stones may also form in persons who have chronic bowel inflammation or who have undergone intestinal bypass or ostomy surgery. As mentioned previously, struvite stones can form in persons who have had a urinary tract infection. Finally, persons who receive the protease inhibitor indinavir are at risk of having kidney stones.
KIDNEY STONES
Kidney stones may contain various combinations of chemicals. The most common type of stone contains calcium in combination with either oxalate or phosphate. Other types of stones include struvite or infection stones, uric acid stones, and cystine stones. Kidney stones may be as small as a grain of sand or as large as a golf ball. They may be smooth or jagged and are usually yellow or brown in color.
Urolithiasis is a medical term used to describe stones occurring in the urinary tract. Other frequently used terms are urinary tract stone disease and nephrolithiasis.